Obesity, metabolic syndrome, and adipocytes

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dc.contributor.authorBergen, Werner G.
dc.contributor.authorFernyhough, Melinda E.
dc.contributor.authorHausman, Gary J.
dc.contributor.authorDodson, Mike V.
dc.contributor.authorDu, Min
dc.contributor.authorMir, Priya S.
dc.contributor.authorGuan, Leluo
dc.contributor.authorJiang, Zhihua
dc.date.accessioned2025-05-01T11:54:40Z
dc.date.available2025-05-01T11:54:40Z
dc.date.issued2011
dc.descriptionObesity and metabolic syndromes are examples whereby excess energy consumption and energy flux disruptions are causative agents of increased fatness. Because other, as yet elucidated, cellular factors may be involved and because potential treatments of these metabolic problems involve systemic agents that are not adipose depot-specific in their actions, should we be thinking of adipose depot-specific (cellular) treatments for these problems? For sure, whether treating obesity or metabolic syndrome, the characteristics of all adipose depot-specific adipocytes and stromal vascular cells should be considered. The focus of this paper is to begin to align metabolic dysfunctions with specific characteristics of adipocytes.
dc.identifier.doihttps://doi.org/10.7939/R3KK94R8S
dc.language.isoen
dc.relationhttp://dx.doi.org/10.1155/2011/721686
dc.relation.isversionofDodson, M. V., Mir, P. S., Hausman, G. J., Guan, L. L., Du, M., Jiang, Z., Fernyhough, M. E., & Bergen, W. G. (2011). Obesity, metabolic syndrome, and adipocytes. Journal of Lipids, 2011(721686), [5 pages]. http://dx.doi.org/10.1155/2011/721686
dc.rightsAttribution 4.0 International
dc.subjectBody Weight
dc.subjectEnergy Consumption
dc.subjectForce & Energy
dc.subjectMetabolic Syndrome--Treatment
dc.subjectFat Cells
dc.subjectObesity
dc.titleObesity, metabolic syndrome, and adipocytes
dc.typehttp://purl.org/coar/resource_type/c_6501 http://purl.org/coar/version/c_970fb48d4fbd8a85
ual.jupiterAccesshttp://terms.library.ualberta.ca/public

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